What is the difference between osa and csa




















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Two Types of Sleep Apnea There are two main types of sleep apnea. Diagnosing the Difference Between Obstructive and Central Apnea The two types of sleep apnea can be difficult to diagnose because their symptoms often overlap. Check our our guide:. A definitive diagnosis of CSA is made using an in-lab polysomnography, which is a detailed sleep study that measures breathing, respiratory effort, electrocardiogram, heart rate, oxygen, eye movement activity, muscle activity, and electrical activity of the brain during an overnight stay in a sleep clinic.

Because central sleep apnea can be tied to several health problems, a healthcare provider may also recommend other tests, such as a brain scan or an echocardiogram of the heart to determine the underlying cause. Anyone who has noticed potential symptoms of central sleep apnea should speak with a doctor who can review their situation and determine if any diagnostic testing is appropriate.

The key to treating central sleep apnea is addressing any underlying health issues that are causing the condition. The type of treatment for central sleep apnea depends on the category and subtype of central sleep apnea. For example, steps may be taken to mitigate congestive heart failure. Those on opioids or other respiratory-depression medications may gradually reduce and taper off the medications.

If at high altitude, the individual can trek back to sea level. In many cases, focusing on the coexisting problem can relieve or eliminate abnormal breathing during sleep. Supplemental oxygen may be used in a similar way. This treatment has shown promise in improving breathing and sleep quality in some research studies. A healthcare provider with a specialty in sleep medicine would be best to review the benefits and side effects of various treatment options for central sleep apnea.

Eric Suni has over a decade of experience as a science writer and was previously an information specialist for the National Cancer Institute. Truong is a Stanford-trained sleep physician with board certifications in sleep and internal medicine. She is the founder of Earlybird Health. Learn why this may be better for…. Sleep apnea headaches are a type of morning headache common in people with obstructive sleep apnea.

Learn more about what…. An actigraphy device tracks your movements so your doctor can analyze your sleep patterns. Necessary cookies are absolutely essential for the website to function properly. This category only includes cookies that ensures basic functionalities and security features of the website. These cookies do not store any personal information.

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It is mandatory to procure user consent prior to running these cookies on your website. The Sleep Foundation editorial team is dedicated to providing content that meets the highest standards for accuracy and objectivity. Our editors and medical experts rigorously evaluate every article and guide to ensure the information is factual, up-to-date, and free of bias. Updated July 9, Written by Eric Suni. Medically Reviewed by Kimberly Truong.

Then when your patient's breathing is stable, the device puts out just enough pressure support to give an approximate 50 percent reduction in the patient's efforts to breathe, making the device comfortable for the patient. The device activates automatically each night to send signals to the breathing muscle diaphragm via the phrenic nerve to restore a normal breathing pattern.

It monitors respiratory signals while you sleep and helps restore normal breathing patterns. Because the device is implantable and activates automatically, it does not require wearing a mask; however, as with any implantable device procedure, there is a risk of implant site infection.

Because of the implementation of his best practices of Implementing Inbound Marketing in its Medical Practice, he turned the once stagnant online presence of Alaska Sleep Clinic to that of "The Most Trafficked Sleep Center Website in the World" in just 18 months time.

He is the President and CEO of inboundMed and enjoys helping sleep centers across the globe grow their business through his unique vision and experience of over 27 years in sleep medicine. Privacy Policy. Categories Follow Us Subscribe. Discover the essential steps to defining, scoring, and treating central hypopneas. Obstructive Sleep Apnea What is it? There are other benefits of weight loss in patients with sleep disordered breathing SDB , including: Lowered blood pressure Decreased RDI Improved snoring and sleep structure Improved arterial blood gas values and pulmonary function Potential reduction of required optimum continuous positive airway pressure CPAP pressure Weight gain is a prominent source of OSA relapse after surgical treatment.

Central Sleep Apnea What is it? Hypopneas - Obstructive and Central As a sleep technologist, you may be very familiar with central apneas, obstructive apneas, and mixed apneas. Obstructive Hypopneas You can think of obstructive hypopneas as if you're covering a vacuum cleaner's suction nozzle with your hand. Central Hypopneas With central hypopneas, however, it's more like you're using less electricity to run the vacuum cleaner.

It's an obstructive hypopnea if you experience: An increase in PAP flow signal or the flattening of nasal pressure flow Snoring during the event Paradoxical breathing It's only central hypopnea if you're experiencing none of the above. How to Properly Score Each Type of Apnea The distinction between central and obstructive hypopneas got lost somewhere along the line, and labs started to score and report them as a single entity.

CPAP therapy uses a CPAP device that includes: A mask that covers your patient's mouth and nose, a mask that only covers their nose, or prongs you fit into their nose. A tube connecting the mask to the CPAP device's motor.

A motor for blowing air into the tube. Oral appliances are recommended for patients who have mild to moderate OSA. Key Takeaways Two main types of sleep apnea include obstructive sleep apnea most common and central sleep apnea. OSA is where your upper airway gets partially or completely blocked while you sleep. Central sleep apnea CSA , cessation of respiratory drive results in a lack of respiratory movements.

Hypopneas aren't necessarily apneas where you stop breathing completely. This method uses autocorrelation to separate OAs from CAs based on their periodicity. The average of the movement image was then calculated for each event, leading to a 1-dimensional movement signal m t. A Butterworth band-pass filter with a lower cutoff frequency of 0.

Autocorrelation was computed for the filtered signals, and its first 10 peaks if peaks did not exist, 0 was considered were used to train three different binary classifiers to distinguish between OA and CA. Classifiers compared were linear support-vector machines, logistic regression, and random forest. Sample autocorrelation signals with their detected peaks are illustrated in Figure 3 for a CA and an OA.

Autocorrelation signal of the movement in an obstructive apnea OA and central apnea CA. OAs are more periodic due to the existence of breathing effort as compared with CAs. Therefore, the OA autocorrelation signal has more peaks, as indicated by red stars. CA: central apnea; OA: obstructive apnea. Lag L was set equal to the event duration. The summations are over all the values of t. This method separates OAs from CAs based on their range of motion.

Histogram of the movement magnitude was constructed for movements in the range of 0 to 0. The average of each bins features of histogram across an event was computed. Principal component analysis PCA was subsequently applied to reduce the number of bins.

Histogram of movement magnitudes. Obstructive apneas have more range of motion as compared with central apneas because of the breathing effort. This method separates OAs from CAs based on the frequency-domain representation of movement histograms. The data were divided into training and validation and test sets by the room in which the study was conducted to ensure the setup camera placement did not affect the algorithm performance.

The recorded data of 21 participants recorded in laboratory room number 1 were used in the training and validation sets. This set included 40 CAs and OAs. The remaining 21 recorded in laboratory room number 2 comprised the test set, which included 75 CAs and OAs. For the autocorrelation and movement histogram methods, classifier hyperparameters were tuned via 3-fold cross-validation on the training set.

Performance of the head, chest, and abdomen bounding box detection was evaluated based on accuracy at the intersection over union values higher than 0. Data from 42 participants 27 men and 15 women were collected for this study. None of the parameters were significantly different between the groups, except BMI with a P value of. BMI is different between the two rooms with a P value of. Figure 6 shows a sample image frame, as well as the manually marked and automatically detected bounding boxes, for the chest and the abdomen.

The performance of the head, chest, and abdomen bounding box detection is quantified in Table 3. Sample chest, abdomen, and head detection results.

Manually annotated and detected regions are shown in blue hashed line and orange solid line, respectively. Different classifiers obtained similar performance for the movement histograms method. For the sake of space, only results of the random forest classifier are shown in Table 4.

The 3D-CNN model obtained the best performance with The proposed 3D-CNN model outperformed all three baseline methods. We hypothesized that localizing the chest and abdomen in the video will increase the signal-to-noise ratio to improve performance. However, as shown in Table 4 , applying 3D-CNN on the whole image frame obtained the best performance.

Figure 7 illustrates how the use of a blanket may explain these results. The blanket propagates chest and abdominal movements outside of their respective detected or annotated regions. As a result, localizing the chest and abdomen locations removed part of the respiratory-related movement signal. Attention mechanisms could also potentially be used to automatically identify image regions in which chest or abdomen movements are prominent.

Annotated chest and abdomen regions do not capture a large area where most of the respiratory-related movement is visible. Manually annotated chest and abdomen regions are shown with blue boxes. Areas with large movement intensity magnitude of the optical flow are highlighted in pink. The methods developed and evaluated in this study are the first to use computer vision to differentiate between OAs and CAs. Although the models were externally validated on data collected in a different room, a limitation of this study was that the same camera model and camera setup was used to collect data in both rooms.

Remedying this limitation will involve external validation on a dataset that will be recorded under different conditions, for example, use of another camera model, different viewing angle, or a different camera distance from the bed. This will evaluate how the models trained here generalize to potential variations that might occur in real-life scenarios, for example, in the home.

Another limitation of the current system is that it relies on the assumption that apneas were already segmented, for example, using previously developed vision-based methods [ 21 ]. Remedying this limitation will involve evaluation of how the combination of such methods with the models developed here will perform.

To address the challenges associated with PSG, there have been several investigations to develop convenient sleep apnea screening devices that can also distinguish CA from OA [ 14 , 15 , 28 , 31 , 32 ].

In a study proposed by Argod et al [ 15 ], pulse transit time technique was used to measure the delay between the R-wave on the electrocardiogram and a finger. They used the delay to visually classify CA from OA [ 15 ]. In another study, Park et al [ 31 ] designed an invasive implantable cardiac device to differentiate between CA and OA based on oscillation characteristics of the cardiac electrical activity. Luo et al [ 14 ] used the diaphragm electromyogram to track the activity of respiratory muscles to differentiate OA from CA.

Thomas et al [ 28 ] used a single-lead electrocardiogram to classify OA from CA by measuring the elevated low-frequency coupling of heart rate variability and the fluctuations in the amplitude of the R-wave.



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